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Shingles: More Than Skin Deep, Linked to Heart Risks
30 Sep
Summary
- Shingles may increase the risk of stroke and heart attack.
- The varicella-zoster virus causes both chickenpox and shingles.
- Evidence is strongest for stroke risk soon after shingles.
Shingles, a condition characterized by a painful rash and nerve pain, is now understood to have implications beyond the skin and nerves. Emerging research indicates a correlation between shingles and an elevated risk of stroke, heart attack, and other cardiovascular events.
The varicella-zoster virus is the causative agent for both chickenpox and shingles. Following an initial chickenpox infection, the virus can lie dormant in nerve cells. It can later reactivate, traveling along a nerve to manifest as shingles, typically presenting as a blistering rash on one side of the body or face.
Evidence particularly highlights a heightened risk of stroke, especially in the immediate aftermath of a shingles episode. This underscores the importance of understanding shingles not just as a dermatological issue but as a viral illness with potential systemic vascular complications.